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Lipotropic
agents are a class of substances that play an
important role in the body's use of fat. Many substances have
lipotropic properties of which choline, inositol and methionine are
among the most noteworthy. Through their involvement in lipid
(fat) metabolism, lipotropics help maintain a healthy
liver.
Choline
& Inositol are co-enzymes that are required
for the proper metabolism of fats, and have the ability to remove
fat from the liver. Choline's function in fat metabolism is
tied to its role in bile production. Without choline, fats can
become trapped in the liver, where they can block normal metabolic
functions. Choline and inositol are necessary for normal nerve
and brain function. Choline is a fat
emulsifier which aids in emulsifying cholesterol so that it doesn't
settle on arterial walls. Choline works well
with inositol to utilize fats and cholesterol. The body can produce
choline, with the help of vitamin B12, folic acid and
methionine. Natural choline production however, may not always
be adequate to meet daily needs. Studies show that diets
deficient in choline lead to undesirable changes to liver, kidney
and brain functions.
Inositol
metabolizes fats and cholesterol and aids in transporting fat in the
blood system. Thus, inositol is an aid in the
redistribution of body fat and can help to lower cholesterol
levels. It is a member of the B-Complex group and is a
lipotropic agent. A lack of inositol has be shown to
produce an accumulation of triglycerides in the
liver.
Methionine has lipotropic properties
similar to those of choline. Two other important amino acids,
cysteine and taurine, depend on adequate levels of methionine for
their biosynthesis in the body. Methionine is one of the
nutrients required during the body's manufacture of choline and
a deficiency of this amino acid will adversely affect fat
metabolism.
Methionine is
a lipotropic amino acid which reduces fat and aids in lowering
cholesterol. It can be substituted for
choline, which aids in reducing liver fat and detoxifies amines,
which are by-products of protein metabolism. A deficiency may
lead to fatty degeneration and cirrhosis of the
liver.
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References
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